What if, suddenly, the army of cells that protect you consider that the colleagues that make your skin, your muscles or even your brain are the enemy to abate?
This is the kind of self-sabotage that support people with autoimmune diseases, a failed response of our body's defenses.
According to the latest data from the World Health Organization (WHO), today more than eighty complaints of this type, affecting between 3% and 7% of Western populations are known. Some are well known, such as diabetes, celiaquismo, Crohn's disease or systemic lupus erythematosus.
Others, such as idiopathic thrombocytopenic purpura or Behçet's syndrome have been classified as rare diseases. And although there are many treatment has no definite cure for now.
What is worrying about these evils is still ignore most of its biological mechanisms. As Kenneth Michael Pollard, the Scripps Research Institute in California, "for every immunologist who you ask, you'll get one or two different theories."
Many foundations of these disorders are considered separately, but there is no common explanation of its origin is successful.
What is clear is that, in most cases, autoimmunity occurs when proteins called autoantibodies designated as hostile to apparently healthy cells. This is what happens with Graves' disease, in which autoantibodies bind to hormone receptors and thyroid excess activated.
This causes hyperthyroidism, running weight loss, bulging eyes, muscle weakness and tremors, among other symptoms. This also explains the immune anarchy rheumatoid arthritis triggered when forming the joint cartilage is destroyed by an antibody called rheumatoid factor; and type 1 diabetes, in which the immune system attacks the insulin-producing cells of the pancreas.
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